Terkko Navigator / Suomalainen, M.

Probiotic supplementation restores normal microbiota composition and function in antibiotic-treated and in caesarean-born infants.

Human single-chain urokinase is activated by the omptins PgtE of Salmonella enterica and Pla of Yersinia pestis despite mutations of active site residues.

Tethered virions are intermediates in the assembly and release of HIV-1 particles.

Notch signalling is required for the survival of epithelial stem cells in the continuously growing mouse incisor.

Splitting placodes: effects of bone morphogenetic protein and Activin on the patterning and identity of mouse incisors.

Temperature-Induced Changes in the Lipopolysaccharide of Yersinia pestis Affect Plasminogen Activation by the Pla Surface Protease.

Patterns of Wnt pathway activity in the mouse incisor indicate absence of Wnt/beta-catenin signaling in the epithelial stem cells.

Invited review: Breaking barriers -- attack on innate immune defences by omptin surface proteases of enterobacterial pathogens.

Using every trick in the book: the Pla surface protease of Yersinia pestis.

The Vpu-regulated endocytosis of HIV-1 Gag is clathrin-independent.

Affecting tooth morphology and renewal by fine-tuning the signals mediating cell and tissue interactions.

Regulation of epithelial stem cells in tooth regeneration.

An integrated gene regulatory network controls stem cell proliferation in teeth.

Vpu and Tsg101 regulate intracellular targeting of the human immunodeficiency virus type 1 core protein precursor Pr55gag.

Meeting report: EMBO workshop 'Cell Biology of Virus Infection', September 25-29, 2004, EMBL, Heidelberg, Germany.

Follistatin regulates enamel patterning in mouse incisors by asymmetrically inhibiting BMP signaling and ameloblast differentiation.

Modulation of activin/bone morphogenetic protein signaling by follistatin is required for the morphogenesis of mouse molar teeth.

Lack of O-antigen is essential for plasminogen activation by Yersinia pestis and Salmonella enterica.

Yersinia pestis Pla has multiple virulence-associated functions.

Human immunodeficiency virus type 1 assembly and lipid rafts: Pr55(gag) associates with membrane domains that are largely resistant to Brij98 but sensitive to Triton X-100.

Lipid rafts and assembly of enveloped viruses.

Meeting report from the EMBO workshop "The Cell Biology of Virus Infection", Heidelberg, Germany, 22-26 September 2001.

Protein regions important for plasminogen activation and inactivation of alpha2-antiplasmin in the surface protease Pla of Yersinia pestis.

Adenovirus-activated PKA and p38/MAPK pathways boost microtubule-mediated nuclear targeting of virus.

The first step of adenovirus type 2 disassembly occurs at the cell surface, independently of endocytosis and escape to the cytosol.

Microtubule-dependent plus- and minus end-directed motilities are competing processes for nuclear targeting of adenovirus.

Lack of effect of leptin on the behaviour of mice predicting the level of anxiety and depression.

The role of the nuclear pore complex in adenovirus DNA entry.

Activation of pro-matrix metalloproteinase-9 and degradation of gelatin by the surface protease PgtE of Salmonella enterica serovar Typhimurium.

Drifting Motions of the Adenovirus Receptor CAR and Immobile Integrins Initiate Virus Uncoating and Membrane Lytic Protein Exposure.

Regulation of a viral proteinase by a peptide and DNA in one-dimensional space. I. binding to DNA and to hexon of the precursor to protein VI, pVI, of human adenovirus.

A direct and versatile assay measuring membrane penetration of adenovirus in single cells.

Tracking viral genomes in host cells at single-molecule resolution.

Chemical Induction of Unfolded Protein Response Enhances Cancer Cell Killing through Lytic Virus Infection.

Co-option of Membrane Wounding Enables Virus Penetration into Cells.

Fluorescence Tracking of Genome Release during the Mechanical Unpacking of Single Viruses.

Plaque2.0-A High-Throughput Analysis Framework to Score Virus-Cell Transmission and Clonal Cell Expansion.

The cell cycle-dependent kinase Cdk9 is a post-exposure drug target against human adenoviruses.

The adenovirus major core protein VII is dispensable for virion assembly but is essential for lytic infection.

Key Role of the Scavenger Receptor MARCO in Mediating Adenovirus Infection and Subsequent Innate Responses of Macrophages.

Erratum for Maler et al., "Key Role of the Scavenger Receptor MARCO in Mediating Adenovirus Infection and Subsequent Innate Responses of Macrophages".

Adenoviral vector with shield and adapter increases tumor specificity and escapes liver and immune control.

Lung macrophage scavenger receptor SR-A6 (MARCO) is an adenovirus type-specific virus entry receptor.

Single entity resolution valving of nanoscopic species in liquids.