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Type XVIII collagen degradation products in acute lung injury.

Collagen XVIII/endostatin is associated with the epithelial-mesenchymal transformation in the atrioventricular valves during cardiac development.

Collagen XVIII/endostatin shows a ubiquitous distribution in human ocular tissues and endostatin-containing fragments accumulate in ocular fluid samples.

Collagen XVIII modulation is altered during progression of oral dysplasia and carcinoma.

Collagen XVIII modulation is altered during progression of oral dysplasia and carcinoma.

Collagen XVIII Short Isoform Is Critical for Retinal Vascularization, and Overexpression of the Tsp-1 Domain Affects Eye Growth and Cataract Formation.

Collagen XVIII in tissue homeostasis and dysregulation - Lessons learned from model organisms and human patients.

Collagen XVIII Deposition in the Basement Membrane Zone beneath the Newly Forming Epidermis during Wound Healing in Mice.

Identification of L-selectin binding heparan sulfates attached to collagen type XVIII.

Basement Membrane Zone Collagens XV and XVIII/Proteoglycans Mediate Leukocyte Influx in Renal Ischemia/Reperfusion.

Lack of collagen XVIII long isoforms affects kidney podocytes while the short form is needed in the proximal tubular basement membrane.

Collagens xv and XVIII show different expression and localisation in cutaneous squamous cell carcinoma: type XV appears in tumour stroma while XVIII becomes up-regulated in tumour cells and lost from microvessels.

Collagens xv and XVIII show different expression and localisation in cutaneous squamous cell carcinoma: type XV appears in tumour stroma while XVIII becomes up-regulated in tumour cells and lost from microvessels.

Lack of collagen XVIII leads to lipodystrophy and perturbs hepatic glucose and lipid homeostasis.

Lack of collagen XVIII leads to lipodystrophy and perturbs hepatic glucose and lipid homeostasis.

The Multiple Fuctions of Collagen Xviii in Development and Disease.

The N-terminal Domain of Unknown Function (DUF959) in Collagen XVIII is Intrinsically Disordered and Highly O-glycosylated.

The N-terminal Domain of Unknown Function (DUF959) in Collagen XVIII is Intrinsically Disordered and Highly O-glycosylated.

The N-terminal Domain of Unknown Function (DUF959) in Collagen XVIII is Intrinsically Disordered and Highly O-glycosylated.

Collagens XV and XVIII show different expression and localisation in cutaneous squamous cell carcinoma: type XV appears in tumor stroma, while XVIII becomes upregulated in tumor cells and lost from microvessels

Differential Expression of Collagen Types XVIII/Endostatin and XV in Normal, Keratoconus, and Scarred Human Corneas.

Differential expression of collagen types XVIII/endostatin and XV in normal, keratoconus, and scarred human corneas.

A Cryptic Frizzled Module in Cell Surface Collagen 18 Inhibits Wnt/beta-Catenin Signaling.

Lack of collagen XVIII accelerates cutaneous wound healing, while overexpression of its endostatin domain leads to delayed healing.

Endostatin Affects Osteoblast Behavior In Vitro, but Collagen XVIII/Endostatin Is Not Essential for Skeletal Development In Vivo.

Endostatin Affects Osteoblast Behavior In Vitro, but Collagen XVIII/Endostatin Is Not Essential for Skeletal Development In Vivo.

Expression of VEGF and collagen XVIII in meningiomas: correlations with histopathological and MRI characteristics.

Expression of VEGF and collagen XVIII in meningiomas: correlations with histopathological and MRI characteristics.

Image based assessment of microvascular function and structure in Collagen XV and XVIII deficient mice.

Specific collagen XVIII isoforms promote adipose tissue accrual via mechanisms determining adipocyte number and affect fat deposition.

Specific collagen XVIII isoforms promote adipose tissue accrual via mechanisms determining adipocyte number and affect fat deposition.

Specific collagen XVIII isoforms promote adipose tissue accrual via mechanisms determining adipocyte number and affect fat deposition.

Absence of collagen XVIII in mice causes age-related insufficiency in retinal pigment epithelium proteostasis.

Differential expression of collagen types XVIII/endostatin and XV in normal, keratoconus, and scarred human corneas

Heparan sulfate of perlecan is involved in glomerular filtration.

Endostatin overexpression specifically in the lens and skin leads to cataract and ultrastructural alterations in basement membranes.

Novel pathogenic mutations and skin biopsy analysis in Knobloch syndrome.

Endostatin, an Inhibitor of Angiogenesis, Decreases After Bidirectional Superior Cavopulmonary Anastamosis.

Comprehensive RNA sequencing in primary murine keratinocytes and fibroblasts identifies novel biomarkers and provides potential therapeutic targets for skin-related diseases

Cytoplasmic polyadenylation by TENT5A is required for proper bone formation

Identification of an Altered Matrix Signature in Kidney Aging and Disease

Discoidin domain receptor regulates ensheathment, survival, and caliber of peripheral axons

Moderate antiangiogenic activity by local, transgenic expression of endostatin in Rip1Tag2 transgenic mice.

Subendothelial Heparan Sulfate Proteoglycans Become Major L-Selectin and Monocyte Chemoattractant Protein-1 Ligands upon Renal Ischemia/Reperfusion.

Subendothelial Heparan Sulfate Proteoglycans Become Major L-Selectin and Monocyte Chemoattractant Protein-1 Ligands upon Renal Ischemia/Reperfusion.

Subendothelial Heparan Sulfate Proteoglycans Become Major L-Selectin and Monocyte Chemoattractant Protein-1 Ligands upon Renal Ischemia/Reperfusion.

Subendothelial Heparan Sulfate Proteoglycans Become Major L-Selectin and Monocyte Chemoattractant Protein-1 Ligands upon Renal Ischemia/Reperfusion.

Endostatin signaling and regulation of endothelial cell-matrix interactions

Endostatin inhibits VEGF-A induced osteoclastic bone resorption in vitro.

Endostatin overexpression inhibits lymphangiogenesis and lymph node metastasis in mice.