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Complement C3a and C3a Receptor Activation Mediates Podocyte Injuries in the Mechanism of Primary Membranous Nephropathy

Cells, Vol. 11, Pages 2103: mTOR-Dependent Autophagy Regulates Slit Diaphragm Density in Podocyte-like Drosophila Nephrocytes

IJMS, Vol. 23, Pages 7276: Potential Renal Damage Biomarkers in Alport Syndrome—A Review of the Literature

Tangshenning Attenuates High Glucose-Induced Podocyte Injury via Restoring Autophagy Activity through Inhibiting mTORC1 Activation

Tangshenning Attenuates High Glucose-Induced Podocyte Injury via Restoring Autophagy Activity through Inhibiting mTORC1 Activation

Identification of Circular Dorsal Ruffles as Signal Platforms for the AKT pathways in Glomerular Podocytes

Distinct APOL1 functions in trypanosomes and kidney podocytes

Zhen-Wu-Tang Induced Mitophagy to Protect Mitochondrial Function in Chronic Glomerulonephritis via PI3K/AKT/mTOR and AMPK Pathways

Flexible and multifaceted: the plasticity of renin-expressing cells

Nephroprotective Effect of Leu-Ile-Lys Tripeptide in Experimental Diabetes Mellitus

Podocyte-associated proteins FAT, alpha-actinin-4 and filtrin are expressed in Langerhans islets of the pancreas.

ARHGEF7 (β-PIX) Is Required for the Maintenance of Podocyte Architecture and Glomerular Function.

Podocyte EGFR Inhibits Autophagy Through Upregulation of Rubicon in Type II Diabetic Nephropathy

Podocyte EGFR Inhibits Autophagy Through Upregulation of Rubicon in Type II Diabetic Nephropathy

Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway.

Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway.

Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway.

Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway

Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway

Podocyte Cell-Specific Expression of Doxycycline Inducible Cre Recombinase in Mice.

Podocyte Autophagy in Homeostasis and Disease

Podocyte protection by Angptl3 knockout via inhibiting ROS/GRP78 pathway in LPS-induced acute kidney injury

Podocyte Aging: Why and How Getting Old Matters

Podocyte Aging: Why and How Getting Old Matters

Podocyte Injury in Autosomal Dominant Polycystic Kidney Disease.

Tacrolimus Protects Podocytes from Apoptosis via Downregulation of TRPC6 in Diabetic Nephropathy

Tacrolimus Protects Podocytes from Apoptosis via Downregulation of TRPC6 in Diabetic Nephropathy

Mitoquinone Protects Podocytes from Angiotensin II-Induced Mitochondrial Dysfunction and Injury via the Keap1-Nrf2 Signaling Pathway

Klotho Ameliorates Podocyte Injury through Targeting TRPC6 Channel in Diabetic Nephropathy

Klotho Ameliorates Podocyte Injury through Targeting TRPC6 Channel in Diabetic Nephropathy

Proteolytic cleavage of Podocin by Matriptase exacerbates podocyte injury [Cell Biology]

Gandi Capsule Improved Podocyte Lipid Metabolism of Diabetic Nephropathy Mice through SIRT1/AMPK/HNF4A Pathway

Twist1 in podocytes ameliorates podocyte injury and proteinuria by limiting CCL2-dependent macrophage infiltration

Proteome Analysis of Isolated Podocytes Reveals Stress Responses in Glomerular Sclerosis.

Tangeretin Ameliorates Glucose-Induced Podocyte Injury through Blocking Epithelial to Mesenchymal Transition Caused by Oxidative Stress and Hypoxia.

Autonomous Calcium Signaling in Human and Zebrafish Podocytes Controls Kidney Filtration Barrier Morphogenesis

Evaluation of podocyte Rac-1 induced kidney disease by modulation of TRPC5

Loss of the Podocyte-Expressed Transcription Factor Tcf21/Pod1 Results in Podocyte Differentiation Defects and FSGS.

Accumulation of Globotriaosylceramide in Podocytes in Fabry Nephropathy Is Associated with Progressive Podocyte Loss.

Inhibition of miRNA-155 Alleviates High Glucose-Induced Podocyte Inflammation by Targeting SIRT1 in Diabetic Mice

Inhibition of miRNA-155 Alleviates High Glucose-Induced Podocyte Inflammation by Targeting SIRT1 in Diabetic Mice

Deiodinase-3 is a thyrostat to regulate podocyte homeostasis

PFKP Activation Ameliorates Foot Process Fusion in Podocytes in Diabetic Kidney Disease

MicroRNA-466o-3p mediatesβ-catenin-induced podocyte injury by targeting Wilms tumor 1.

Transcriptome analysis of primary podocytes reveals novel calcium regulated regulatory networks.

Islet Transplantation Reverses Podocyte Injury in Diabetic Nephropathy or Induced by High Glucose via Inhibiting RhoA/ROCK/NF-κB Signaling Pathway

Islet Transplantation Reverses Podocyte Injury in Diabetic Nephropathy or Induced by High Glucose via Inhibiting RhoA/ROCK/NF-κB Signaling Pathway

The role of filamins in mechanically stressed podocytes

Death-associated protein kinase 1 correlates with podocyte apoptosis and renal damage and can be mediated by miR-361

Synaptopodin Is Dispensable for Normal Podocyte Homeostasis but Is Protective in the Context of Acute Podocyte Injury.