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Nephroprotective Effect of Leu-Ile-Lys Tripeptide in Experimental Diabetes Mellitus

Identification of Circular Dorsal Ruffles as Signal Platforms for the AKT pathways in Glomerular Podocytes

Flexible and multifaceted: the plasticity of renin-expressing cells

Distinct APOL1 functions in trypanosomes and kidney podocytes

B7-1 mediates podocyte injury and glomerulosclerosis through communication with Hsp90ab1-LRP5-β-catenin pathway

Isolated steroid-resistant nephrotic syndrome in a Chinese child carrying a de novo mutation in WT1 gene:a case report and literature review

Increased severity of chronic kidney disease in response to high potassium intake is dependent on mineralocorticoid receptor activation

Podocyte-associated proteins FAT, alpha-actinin-4 and filtrin are expressed in Langerhans islets of the pancreas.

ARHGEF7 (β-PIX) Is Required for the Maintenance of Podocyte Architecture and Glomerular Function.

Podocyte EGFR Inhibits Autophagy Through Upregulation of Rubicon in Type II Diabetic Nephropathy

Podocyte EGFR Inhibits Autophagy Through Upregulation of Rubicon in Type II Diabetic Nephropathy

Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway.

Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway.

Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway.

Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway

Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway

Podocyte Cell-Specific Expression of Doxycycline Inducible Cre Recombinase in Mice.

Podocyte Autophagy in Homeostasis and Disease

Podocyte protection by Angptl3 knockout via inhibiting ROS/GRP78 pathway in LPS-induced acute kidney injury

Podocyte Aging: Why and How Getting Old Matters

Podocyte Aging: Why and How Getting Old Matters

Podocyte Injury in Autosomal Dominant Polycystic Kidney Disease.

Tacrolimus Protects Podocytes from Apoptosis via Downregulation of TRPC6 in Diabetic Nephropathy

Tacrolimus Protects Podocytes from Apoptosis via Downregulation of TRPC6 in Diabetic Nephropathy

Mitoquinone Protects Podocytes from Angiotensin II-Induced Mitochondrial Dysfunction and Injury via the Keap1-Nrf2 Signaling Pathway

Klotho Ameliorates Podocyte Injury through Targeting TRPC6 Channel in Diabetic Nephropathy

Klotho Ameliorates Podocyte Injury through Targeting TRPC6 Channel in Diabetic Nephropathy

Proteolytic cleavage of Podocin by Matriptase exacerbates podocyte injury [Cell Biology]

Gandi Capsule Improved Podocyte Lipid Metabolism of Diabetic Nephropathy Mice through SIRT1/AMPK/HNF4A Pathway

Twist1 in podocytes ameliorates podocyte injury and proteinuria by limiting CCL2-dependent macrophage infiltration

Proteome Analysis of Isolated Podocytes Reveals Stress Responses in Glomerular Sclerosis.

Tangeretin Ameliorates Glucose-Induced Podocyte Injury through Blocking Epithelial to Mesenchymal Transition Caused by Oxidative Stress and Hypoxia.

Autonomous Calcium Signaling in Human and Zebrafish Podocytes Controls Kidney Filtration Barrier Morphogenesis

Evaluation of podocyte Rac-1 induced kidney disease by modulation of TRPC5

Deiodinase-3 is a thyrostat to regulate podocyte homeostasis

Loss of the Podocyte-Expressed Transcription Factor Tcf21/Pod1 Results in Podocyte Differentiation Defects and FSGS.

Accumulation of Globotriaosylceramide in Podocytes in Fabry Nephropathy Is Associated with Progressive Podocyte Loss.

Inhibition of miRNA-155 Alleviates High Glucose-Induced Podocyte Inflammation by Targeting SIRT1 in Diabetic Mice

Inhibition of miRNA-155 Alleviates High Glucose-Induced Podocyte Inflammation by Targeting SIRT1 in Diabetic Mice

PFKP Activation Ameliorates Foot Process Fusion in Podocytes in Diabetic Kidney Disease

MicroRNA-466o-3p mediatesβ-catenin-induced podocyte injury by targeting Wilms tumor 1.

Transcriptome analysis of primary podocytes reveals novel calcium regulated regulatory networks.

Islet Transplantation Reverses Podocyte Injury in Diabetic Nephropathy or Induced by High Glucose via Inhibiting RhoA/ROCK/NF-κB Signaling Pathway

Islet Transplantation Reverses Podocyte Injury in Diabetic Nephropathy or Induced by High Glucose via Inhibiting RhoA/ROCK/NF-κB Signaling Pathway

The role of filamins in mechanically stressed podocytes

Death-associated protein kinase 1 correlates with podocyte apoptosis and renal damage and can be mediated by miR-361

Synaptopodin Is Dispensable for Normal Podocyte Homeostasis but Is Protective in the Context of Acute Podocyte Injury.

Suppressed ER-associated degradation by intraglomerular cross talk between mesangial cells and podocytes causes podocyte injury in diabetic kidney disease.

Protective Role of the M-Sec-Tunneling Nanotube System in Podocytes

Tripterygium glycoside suppresses epithelial‑to‑mesenchymal transition of diabetic kidney disease podocytes by targeting autophagy through the mTOR/Twist1 pathway