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Hypertrophic cardiomyopathy mutations in the pliant and light chain-binding regions of the lever arm of humanβ-cardiac myosin have divergent effects on myosin function

Cancers, Vol. 14, Pages 2288: The Beneficial Role of Physical Exercise on Anthracy-Clines-Induced Cardiotoxicity in Breast Cancer Patients

Cardiac myosin binding protein-C palmitoylation is associated with increased myofilament affinity, reduced myofilament Ca2+ sensitivity and is increased in ischaemic heart failure.

Dilated cardiomyopathy mutation E525K in human beta-cardiac myosin stabilizes the interacting heads motif and super-relaxed state of myosin

Suppression of the Inhibitory Effect of circ_0036176-Translated Myo9a-208 on Cardiac Fibroblast Proliferation by miR-218-5p

Myosin 7b is a regulatory long noncoding RNA (lncMYH7b) in the human heart

Cardiac ventricular myosin and slow skeletal myosin exhibit dissimilar chemo-mechanical properties despite the same myosin heavy chain isoform.

Cardiac myosin filaments are directly regulated by calcium

Myosin waves and a mechanical asymmetry guide the oscillatory migration of Drosophila cardiac progenitors

Cardiac myosin binding protein-C variants in paediatric-onset hypertrophic cardiomyopathy: natural history and clinical outcomes

Hypertrophic cardiomyopathy β-cardiac myosin mutation (P710R) leads to hypercontractility by disrupting super-relaxed state: multiscale measurements and computational modeling

TWO CLASSES OF MYOSIN INHIBITORS, BLEBBISTATIN AND MAVACAMTEN, STABILIZE β-CARDIAC MYOSIN IN DIFFERENT STRUCTURAL AND FUNCTIONAL STATES

Procoagulant activities of skeletal muscle and cardiac myosins require both myosin protein and myosin-associated anionic phospholipids.

Actin-binding compounds, previously discovered by FRET-based high-throughput screening, differentially affect skeletal and cardiac muscle [Methods and Resources]

Variants identify sarcomere inter-protein contacts distinguishing inheritable cardiac muscle diseases

Human hypertrophic cardiomyopathy mutation R712L suppresses the working stroke of cardiac myosin and can be rescued by omecamtiv mecarbil

Preventive training interferes with mRNA-encoding myosin 7 and collagen I expression during pulmonary arterial hypertension

Direct detection of the myosin super-relaxed state and interacting heads motif in solution

Defining Cardiac Cell Populations and Relative Cellular Composition of the Early Fetal Human Heart

Hypoxia increases the release of salmon cardiac peptide (sCP) from the heart of rainbow trout (Oncorhynchus mykiss) under constant mechanical load in vitro.

Novel Mutations Widen the Phenotypic Spectrum of Slow Skeletal/β-Cardiac Myosin (MYH7) Distal Myopathy.

Mechanical function of cardiac fibre bundles is partly protected by exercise in response to diet-induced obesity in rats

IJMS, Vol. 23, Pages 88: Functional and Molecular Characterisation of Heart Failure Progression in Mice and the Role of Myosin Regulatory Light Chains in the Recovery of Cardiac Muscle Function

IJMS, Vol. 23, Pages 1135: Effect of Myosin Isoforms on Cardiac Muscle Twitch of Mice, Rats and Humans

IJMS, Vol. 23, Pages 1220: The Local Environment of Loop Switch 1 Modulates the Rate of ATP-Induced Dissociation of Human Cardiac Actomyosin

Sept7b is required for the subcellular organization of cardiomyocytes and cardiac function in zebrafish.

Under construction: The dynamic assembly, maintenance, and degradation of the cardiac sarcomere.

Small Molecules acting on Myofilaments as Treatments for Heart and Skeletal Muscle Diseases.

<em>Sept7b</em> is required for the subcellular organization of cardiomyocytes and cardiac function in zebrafish

Highly multiplexed quantitative proteomic and phosphoproteomic analyses in vascular smooth muscle cell dedifferentiation.

TROPONIN REVEALED Uncovering the Structure of the Thin Filament On-Off Switch in Striated Muscle.

Impaired muscle morphology in a Drosophila model of myosin storage myopathy was supressed by overexpression of an E3 ubiquitin ligase [RESEARCH ARTICLE]

Angiotensin-(3–7) alleviates isoprenaline-induced cardiac remodeling via attenuating cAMP-PKA and PI3K/Akt signaling pathways

Post-translational modification patterns on β-myosin heavy chain are altered in ischemic and non-ischemic human hearts

Geometric frustration in the myosin superlattice of vertebrate muscle

Post-translational modification patterns onβ-myosin heavy chain are altered in ischemic and non-ischemic human hearts

IJMS, Vol. 22, Pages 5732: Striated Preferentially Expressed Protein Kinase (SPEG) in Muscle Development, Function, and Disease

Interplay of Genotype and Substrate Stiffness in Driving the Hypertrophic Cardiomyopathy Phenotype in iPSC-Micro-Heart Muscle Arrays

Taking flight: an educational primer for use with“A novel mechanism for activation of myosin regulatory light chain by protein kinase C-delta in Drosophila”

Mep1a contributes to Ang II-induced cardiac remodeling by promoting cardiac hypertrophy, fibrosis and inflammation.

Low-dose erythropoietin improves cardiac function in experimental heart failure without increasing haematocrit.

Overexpression of Vascular Endothelial Growth Factor-B in Mouse Heart Alters Cardiac Lipid Metabolism and Induces Myocardial Hypertrophy.

Overexpression of Vascular Endothelial Growth Factor-B in Mouse Heart Alters Cardiac Lipid Metabolism and Induces Myocardial Hypertrophy.

Overexpression of Vascular Endothelial Growth Factor-B in Mouse Heart Alters Cardiac Lipid Metabolism and Induces Myocardial Hypertrophy.

Overexpression of Vascular Endothelial Growth Factor-B in Mouse Heart Alters Cardiac Lipid Metabolism and Induces Myocardial Hypertrophy.

Maternal hyperglycemia leads to fetal cardiac hyperplasia and dysfunction in a rat model.

Maternal hyperglycemia leads to fetal cardiac hyperplasia and dysfunction in a rat model.

Separation of HCM and LQT Cardiac Diseases with Machine Learning of Ca2+ Transient Profiles.

Gaining insight into cellular cardiac physiology using single particle tracking.

Pyrroloquinoline quinone attenuates isoproterenol hydrochloride‑induced cardiac hypertrophy in AC16 cells by inhibiting the NF‑κB signaling pathway.